Somatostatin-14

Hormonal / Inhibitory

Also known as: SRIF-14, SST-14, Somatotropin Release-Inhibiting Factor, Growth Hormone Inhibiting Hormone, GHIH

SomatostatinsResearch phase: Endogenous hormone (well characterized)Regulatory: Endogenous peptide. Not used clinically due to extremely short half-life. Synthetic analogs (octreotide, lanreotide) are FDA-approved.

Mechanism

Somatostatin-14 is the body's primary brake on growth hormone secretion. This 14-amino-acid cyclic peptide is produced in the hypothalamus, GI tract, and pancreas, where it broadly inhibits the release of numerous hormones including GH, TSH, insulin, glucagon, and gastric acid. It is the endogenous form that inspired drugs like octreotide and lanreotide.

Technical detail

Somatostatin-14 (SRIF-14) is an endogenous cyclic tetradecapeptide (Ala-Gly-Cys-Lys-Asn-Phe-Phe-Trp-Lys-Thr-Phe-Thr-Ser-Cys, disulfide bridge Cys3-Cys14) that binds all five somatostatin receptor subtypes (SSTR1-5), all Gi/Go-coupled GPCRs. Signaling includes inhibition of adenylyl cyclase (reducing cAMP), activation of K+ channels (hyperpolarization), inhibition of voltage-gated Ca2+ channels, and activation of protein phosphatases. In the anterior pituitary, it inhibits GH and TSH secretion. In the pancreas, it inhibits insulin and glucagon. In the GI tract, it suppresses gastric acid, gastrin, secretin, CCK, and VIP. Half-life is only 1-3 minutes due to rapid enzymatic degradation.

Evidence